Posts Tagged ‘Alzheimer’s disease’
Science may just be starting to make some sense of this cruelest of diseases.
It used be to the dreaded ‘C’ word; cancer. But now that ‘C’ word has a companion, the dreaded ‘A’ word. The incidence of Alzheimer’s disease seems to be on a terrible rise. Indeed, my wife, Jean, lost her late husband to Alzheimer’s disease. My half-sister back in England is now very ill with the disease. Just chatting to some people here in Payson a few days ago revealed many who had friends or relations suffering.
So a recent item first seen on the website of The Permaculture Research Institute of Australia really jumped off the ‘page’! It was an article by George Monbiot entitled The Mind Thieves. I dropped Mr. Monbiot a quick email requesting permission to republish the article and very promptly received a positive answer. Thank you, Sir.
So before moving to the article, first a little background on George M. From his website, one quickly reads,
I had an unhappy time at university, and I now regret having gone to Oxford, even though the zoology course I took – taught, among others, by Richard Dawkins, Bill Hamilton and John Krebs – was excellent. The culture did not suit me, and when I tried to join in I fell flat on my face, sometimes in a drunken stupor. I enjoyed the holidays more: I worked on farms and as a waterkeeper on the River Kennet. I spent much of the last two years planning my escape. There was only one job I wanted, and it did not yet exist: to make investigative environmental programmes for the BBC.
I’m not going to copy the full ‘About George‘ description but do urge you to pop across to here and read it yourself; George has had, trust me, a fascinating life journey that I suspect is far from over. This is how that About description closes,
Here are some of the things I love: my family and friends, salt marshes, arguments, chalk streams, Russian literature, kayaking among dolphins, diversity of all kinds, rockpools, heritage apples, woods, fishing, swimming in the sea, gazpacho, sprinting up the pitch in ultimate frisbee, ponds and ditches, growing vegetables, insects, pruning, forgotten corners, fossils, goldfinches, etymology, Bill Hicks, ruins, Shakespeare, landscape history, palaeoecology and Father Ted.
Here are some of the things I try to fight: undemocratic power, corruption, deception of the public, environmental destruction, injustice, inequality and the misallocation of resources, waste, denial, the libertarianism which grants freedom to the powerful at the expense of the powerless, undisclosed interests, complacency.
Here is what I fear: other people’s cowardice.
I still see my life as a slightly unhinged adventure whose perpetuation is something of a mystery. I have no idea where it will take me, and no ambitions other than to keep doing what I do. So far it’s been gripping.
The article was first published in the British Guardian newspaper (there’s an online link to it here) as the article mentions below. But I am republishing, in full thanks to George, the copy that appeared on George’s website on the 10th September last, including the references.
The Mind Thieves
September 10th, 2012
The evidence linking Alzheimer’s disease to the food industry is strong and growing.
By George Monbiot, published in the Guardian, 11th September 2012
When you raise the subject of over-eating and obesity, you often see people at their worst. The comment threads discussing these issues reveal a legion of bullies, who appear to delight in other people’s problems.
When alcoholism and drug addiction are discussed, the tone tends to be sympathetic. When obesity is discussed, the conversation is dominated by mockery and blame, though the evidence suggests that it can be driven by similar forms of addiction(1,2,3,4). I suspect that much of this mockery is a coded form of snobbery: the strong association between poor diets and poverty allows people to use this issue as a cipher for something else they want to say, which is less socially acceptable.
But this problem belongs to all of us. Even if you can detach yourself from the suffering caused by diseases arising from bad diets, you will carry the cost, as a growing proportion of the health budget will be used to address them. The cost – measured in both human suffering and money – could be far greater than we imagined. A large body of evidence now suggests that Alzheimer’s is primarily a metabolic disease. Some scientists have gone so far as to rename it. They call it diabetes type 3.
New Scientist carried this story on its cover last week(5): since then I’ve been sitting in the library trying to discover whether it stands up. I’ve now read dozens of papers on the subject, testing my cognitive powers to the limit as I’ve tried to get to grips with brain chemistry. While the story is by no means complete, the evidence so far is compelling.
Around 35 million people suffer from Alzheimer’s disease worldwide(6); current projections, based on the rate at which the population ages, suggest that this will rise to 100 million by 2050(7). But if, as many scientists now believe, it is caused largely by the brain’s impaired response to insulin, the numbers could rise much further. In the US, the percentage of the population with diabetes type 2, which is strongly linked to obesity, has almost trebled in 30 years(8). If Alzheimer’s, or “diabetes type 3”, goes the same way, the potential for human suffering is incalculable.
Insulin is the hormone which prompts the liver, muscles and fat to absorb sugar from the blood. Diabetes 2 is caused by excessive blood glucose, resulting either from a deficiency of insulin produced by the pancreas, or resistance to its signals by the organs which would usually take up the glucose.
The association between Alzheimer’s and diabetes 2 is long-established: type 2 sufferers are two to three times more likely to be struck by this dementia than the general population(9). There are also associations between Alzheimer’s and obesity(10) and Alzheimer’s and metabolic syndrome (a complex of diet-related pathologies)(11).
Researchers first proposed that Alzheimer’s was another form of diabetes in 2005. The authors of the original paper investigated the brains of 54 corpses, 28 of which belonged to people who had died of the disease(12). They found that the levels of both insulin and insulin-like growth factors in the brains of Alzheimer’s patients were sharply reduced by comparison to those in the brains of people who had died of other causes. Levels were lowest in the parts of the brain most affected by the disease.
Their work led them to conclude that insulin and insulin-like growth factor are produced not only in the pancreas but also in the brain. Insulin in the brain has a host of functions: as well as glucose metabolism, it helps to regulate the transmission of signals from one nerve cell to another, and affects their growth, plasticity and survival(13,14).
Experiments conducted since then appear to support the link between diet and dementia(15,16,17,18), and researchers have begun to propose potential mechanisms. In common with all brain chemistry, these tend to be fantastically complex, involving, among other impacts, inflammation, stress caused by oxidation, the accumulation of one kind of brain protein and the transformation of another(19,20,21,22). I would need the next six pages of this paper even to begin to explain them, and would doubtless get it wrong (if you’re interested, please follow the links on my website).
Plenty of research still needs to be done. But if the current indications are correct, Alzheimer’s disease could be another catastrophic impact of the junk food industry, and the worst discovered so far. Our governments, as they are in the face of all our major crises, appear to be incapable of responding.
In this country as in many others, the government’s answer to the multiple disasters caused by the consumption of too much sugar and fat is to call on both companies and consumers to regulate themselves. Before he was replaced by someone even worse, the former health secretary, Andrew Lansley, handed much of the responsibility for improving the nation’s diet to food and drinks companies: a strategy that would work only if they volunteered to abandon much of their business(23,24).
A scarcely-regulated food industry can engineer its products – loading them with fat, salt, sugar and high fructose corn syrup – to bypass the neurological signals which would otherwise prompt people to stop eating(25). It can bombard both adults and children with advertising. It can (as we discovered yesterday) use the freedoms granted to academy schools to sell the chocolate, sweets and fizzy drinks now banned from sale in maintained schools(26). It can kill the only effective system (the traffic light label) for informing people how much fat, sugar and salt their food contains. Then it can turn to the government and blame consumers for eating the products it sells. This is class war: a war against the poor fought by the executive class in government and industry.
We cannot yet state unequivocally that poor diet is a leading cause of Alzheimer’s disease, though we can say that the evidence is strong and growing. But if ever there was a case for the precautionary principle, here it is. It’s not as if we lose anything by eating less rubbish. Averting a possible epidemic of this devastating disease means taking on the bullies: those who mock people for their pathologies and those who spread the pathologies by peddling a lethal diet.
1. Caroline Davis et al, 2011. Evidence that ‘food addiction’ is a valid phenotype of obesity. Appetite Vol. 57, pp711–717. doi:10.1016/j.appet.2011.08.017
2. Paul J. Kenny, November 2011. Common cellular and molecular mechanisms in obesity and drug addiction. Nature Neuroscience, Vol. 12, pp 638-651. doi:10.1038/nrn3105
3. Joseph Frascella et al, 2010. Shared brain vulnerabilities open the way for nonsubstance addictions: Carving addiction
at a new joint? Annals of the New York Academy of Sciences, Vol. 1187, pp294–315. doi: 10.1111/j.1749-6632.2009.05420.x
4. Ashley N. Gearhardt et al, 2010. Can food be addictive? Public health and policy implications. Addiction, 106, 1208–1212. ad. d_3301 1208..1212 doi:10.1111/j.1360-0443.2010.03301.x
5. Bijal Trivedi, 1st September 2012. Eat Your Way to Dementia. New Scientist.
6. Sónia C. Correia et al, 2011. Insulin-resistant brain state: The culprit in sporadic Alzheimer’s disease? Ageing Research Reviews Vol. 10, 264–273. doi:10.1016/j.arr.2011.01.001
7. Fabio Copped`e et al, 2012. Nutrition and Dementia. Current Gerontology and Geriatrics Research, Vol. 2012, pp1-3. doi:10.1155/2012/926082
8. See the graph in Bijal Trivedi, 1st September 2012. Eat Your Way to Dementia. New Scientist.
9. Johanna Zemva and Markus Schubert, September 2011. Central Insulin and Insulin-Like Growth Factor-1 Signaling – Implications for Diabetes Associated Dementia. Current Diabetes Reviews, Vol.7, No.5, pp356-366. doi.org/10.2174/157339911797415594
10. Eg Weili Xu et al, 2011. Midlife overweight and obesity increase late life dementia risk: a population-based twin study. Neurology, Vol. 76, no. 18, pp.1568–1574.
11. M. Vanhanen et al, 2006. Association of metabolic syndrome with Alzheimer disease: A population-based study. Neurology, vol. 67, pp.843–847.
12. Eric Steen et al, 2005. Impaired insulin and insulin-like growth factor expression and signaling mechanisms in Alzheimer’s disease – is this type 3 diabetes?. Journal of Alzheimer’s Disease, Vol. 7, pp.63–80.
13. Konrad Talbot et al, 2012. Demonstrated brain insulin resistance in Alzheimer’s disease patients is associated with IGF-1 resistance, IRS-1 dysregulation, and cognitive decline. The Journal of Clinical Investigation, Vol.122, No.4, pp.1316–1338. doi:10.1172/JCI59903.
14. Naoki Yamamoto et al, 2012. Brain insulin resistance accelerates Aβ fibrillogenesis by inducing GM1 ganglioside clustering in the presynaptic membranes. Journal of Neurochemistry, Vol. 121, 619–628. doi: 10.1111/j.1471-4159.2012.07668.x
Wei-Qin Zhao and Matthew Townsend, 2009. Insulin resistance and amyloidogenesis as common molecular foundation for type 2 diabetes and Alzheimer’s disease.
Biochimica et Biophysica Acta, Vol.1792, pp.482–496. doi.org/10.1016/j.bbadis.2008.10.014,
16. Sónia C. Correia et al, 2011. Insulin-resistant brain state: The culprit in sporadic Alzheimer’s disease? Ageing Research Reviews Vol. 10, 264–273. doi:10.1016/j.arr.2011.01.001
17. T. Ohara et al, 2011. Glucose tolerance status and risk of dementia in the community, the Hisayama study. Neurology, Vol. 77, pp.1126–1134.
18. Karen Neumann et al, 2008. Insulin resistance and Alzheimer’s disease: molecular links & clinical implications. Current Alzheimer Research, Vol.5, no.5, pp438–447.
19. Eg: Lap Ho et al, 2012. Insulin Receptor Expression and Activity in the Brains of Nondiabetic Sporadic Alzheimer’s Disease Cases. International Journal of Alzheimer’s Disease, Volume 2012. doi:10.1155/2012/321280
20. Suzanne M. de la Monte, 2012. Contributions of Brain Insulin Resistance and Deficiency in Amyloid-Related Neurodegeneration in Alzheimer’s Disease. Drugs, Vol. 72, no.1, pp. 49-66. doi: 10.2165/11597760
21. Ying Liu et al, 2011. Deficient brain insulin signalling pathway in Alzheimer’s disease and diabetes. Journal of Pathology, Vol. 225, pp.54–62. doi: 0.1002/path.2912
22. Konrad Talbot et al, 2012. Demonstrated brain insulin resistance in Alzheimer’s disease patients is associated with IGF-1 resistance, IRS-1 dysregulation, and cognitive decline. The Journal of Clinical Investigation, Vol.122, No.4, pp.1316–1338. doi:10.1172/JCI59903.
Don’t know about you but the above is a fine example of investigative reporting. It deserves the widest circulation because if it is proved that there is a link between diet and Alzheimer’s disease then, once again, it shows how taking personal responsibility for our health has huge implications for us, our families and for society at large.
Is there a link between anxiety and dementia?
Before going to a recent BBC report about this important subject, let me offer a personal anecdote.
A couple of months ago I had cause to be seen by a neurologist. I wanted to get a professional opinion as to whether a degree of forgetfulness that I was experiencing was normal for a person of my age (68 next birthday). Dr. G. not only confirmed that there was absolutely no sign of dementia but that my forgetfulness was perfectly normal for someone of my age who had been through some major life changes in the last few years.
Dr. G. stressed (probably not the best word but you know what I mean!) that me worrying about forgetting stuff and the resulting anxiety was a self-feeding issue. I had to stop being anxious. Indeed, Dr. G. said the following (and this I haven’t forgotten!):
Anxiety is the killer of good bodies and the killer of good brains!
So with those words ringing in your ears, have a read of this recent report from the BBC News website.
Role of stress in dementia investigated
By Michelle Roberts, Health editor, BBC News online
UK experts are to begin a study to find out if stress can trigger dementia.
The investigation, funded by the Alzheimer’s Society, will monitor 140 people with mild cognitive impairment or “pre-dementia” and look at how stress affects their condition.
The researchers will take blood and saliva samples at six-monthly intervals over the 18 months of the study to measure biological markers of stress.
They hope their work will reveal ways to prevent dementia.
The results could offer clues to new treatments or better ways of managing the condition, they say.
People who have mild cognitive impairment are at an increased risk of going on to develop dementia – although some will remain stable and others may improve.
And past work suggests mid-life stress may increase a person’s risk of Alzheimer’s disease.
A Swedish study that followed nearly 1,500 women for a period of 35 years found the risk of dementia was about 65% higher in women who reported repeated periods of stress in middle age than in those who did not.
Scottish scientists, who have done studies in animals, believe the link may be down to hormones the body releases in response to stress which interfere with brain function.
Prof Clive Holmes, from the University of Southampton, who will lead the study, said: “All of us go through stressful events. We are looking to understand how these may become a risk factor for the development of Alzheimer’s.
“Something such as bereavement or a traumatic experience – possibly even moving home – are also potential factors.
“This is the first stage in developing ways in which to intervene with psychological or drug-based treatments to fight the disease.
“We are looking at two aspects of stress relief – physical and psychological – and the body’s response to that experience.”
Dr Simon Ridley, of Alzheimer’s Research UK, said: “We welcome any research that could shed new light on Alzheimer’s disease and other causes of dementia.
“Understanding the risk factors for Alzheimer’s could provide one piece of the puzzle we need to take us closer to a treatment that could stop the disease in its tracks.”
Finally, let me leave you with this.
Some of life’s lessons are easy to learn – once we have been taught!
Readers be warned! This is one of my more subjective posts written in the hope that many will ‘connect’ with the emotions expressed.
I want to explore the power of touch.
Not just in a direct manner such as a hug or an arm around the shoulder but also the way that love can reach out and ‘touch’ us from afar. I’m going to do that by recounting something that Jean and I have experienced over the last couple of weeks. Here we go!
A while after we had moved to Payson in February, 2010 both Jean and I noticed that I was getting forgetful. Initially we thought it was just a characteristic of the vestibular migraine that I was diagnosed with in 2009 but eventually it seemed a good idea to have a local doctor here in Payson check me out. That examination took place last April 24th., a little over two weeks ago. The doctor dropped a huge bombshell in our laps by saying that she thought that I was exhibiting signs of early-onset Alzheimer’s Disease!
To say that I was shocked was an understatement. Jean was beside herself with worry as her late American husband, who had died in 2005, had suffered badly from Alzheimer’s Disease for his last two years. We had 48-hours of wall-to-wall worry!
A few days later, I was getting very angry at the lack of gentleness in the way that the doctor had spoken to me. Jean had the brilliant idea of contacting a retired doctor friend. His response was loud and clear; he advised me to get a second opinion and continued by recommending a neurologist that he knew well. That appointment was held this last Tuesday, May 8th, down in Phoenix.
The neurologist asked me many questions, including such verbal tests as how many animals can you name in a minute, spell the word ‘world’ backwards, deduct 7 from 95 (and then keep deducting 7 from the resultant answer!!), and then undertook a physical examination including co-ordination skills, blood flow in the neck and other relevant aspects.
All of which lead him to the conclusion that I was not showing any signs of dementia. My forgetfulness was normal for someone of my age (67 last birthday) especially taking into account all the life changes of the past few years.
Then the neurologist went on to warn me about anxiety. He said it was a ‘killer’ of healthy bodies and healthy minds, especially as we got older. So my anxiety over my sister’s dementia, my half-sister living in Devon, England, whom I am very close to, is now badly affected by vascular dementia and got me thinking I might be following in her path, and my anxiety over thinking my life now was ‘too good to be true’, was getting in the way of me being a relaxed, ‘go with the flow’ individual.
Thus a couple of extremely stressful and worrying weeks came to a most wonderful conclusion; an outcome that couldn’t have been better. The degree of emotional and psychological disruption that Jean and I have been through was not however without some major lessons being learnt.
Being scared – I’ve always taken for granted that I would have good health throughout my life, aided and abetted by the fact that I have never been admitted to hospital and have avoided serious illnesses. The first doctor’s so-called diagnosis was one giant slap-in-the-face especially realising that the future in store could be a steady decline in my cognitive skills.
For the first time in my life I was truly scared and last Monday, the day before the visit to the neurologist, I broke down in Jean’s arms saying how scared I was. Revealing such vulnerability was not easy for me but being held by Jean under those circumstances was deep and pure bliss. As the saying goes, ‘If one doesn’t run the risk of being lost, then one can never be found.’
Love and friendship – The number of people that came up to Jean and me, gave us big hugs and said that they would be thinking of us during our trip down to Phoenix last Tuesday was indescribably beautiful. So many showed such a depth of feeling for what Jean and I were going through. Many others from distant places sent encouraging emails or telephoned. It all amounted to preventing us from feeling alone and reinforced our determination that whatever the medical outcome, we would find a way of handling it.
The power of the mind – my brother-in-law, in a recent telephone call, said that once the mind latches on to an idea, it does everything it can to reinforce that idea, however illogical it may be. Thus over the last couple of weeks, every time I dropped something, or forgot where I had put my glasses, or wasn’t clear which day of the week it was, and on and on, I used that as ‘proof’ that I was rapidly losing my mind. It should serve as a strong warning that we can literally think ourselves into a crisis!
The love between a dog and a human – hugging a dog when one is feeling emotionally vulnerable is beyond measure. Dogs always sense when we humans are feeling fragile and they offer their uncomplicated hearts to us without any condition or need for return. That selfless love is an inspiring example of what we all need to learn to give one another.
Touch and social intimacy – we have so much to learn from dogs when it comes to touch and social intimacy. We are all needy for touch.
Which leads me providentially to a recent item from Terry Hershey. Terry came to Payson in March, 2011 and he was a most inspirational speaker. I have followed him ever since.
Last Monday’s Sabbath Moment included the following story, republished with Terry’s kind permission – the story is all about touch!
Caroline was very sad. Caroline was only six years old and her father had just died. In fact, her father had been assassinated.
Sitting in the back of big black limousine, Caroline Kennedy didn’t quite know what to do with her sadness. On the seat next to her sat her nanny, Maud Shaw, and next to Maud, Caroline’s younger brother John.
Through the windshield Caroline could see her mother, Jackie, and her uncles, Robert and Ted, walking in front of the limousine as it slowly made it’s way down the Boulevard to St. Matthew’s Cathedral. Looking out of her side of the car, Caroline recognized the friendly face of Secret Service agent, Robert Foster. She liked and trusted Robert Foster.
Not knowing what to do with her sadness, and on impulse, she rolled down the window and stuck out her six-year old hand. Agent Foster had a choice to make. Secret Service agents are not allowed to have their hands occupied, needing to be ready for any emergency. But Robert Foster didn’t even think twice. He held Caroline’s hand tightly the entire way to the cathedral.
Later, Agent Foster said it was all he could do to “fight back his own tears of sadness, for little Caroline Kennedy.” When asked about his kindness, he seemed surprised, “All I did was hold a hand,” he answered.
Terry then goes on to say,
We all know sadness. Life breaks for each one of us in different ways and in different places. And sometimes the sadness seems too much to carry.
It requires courage to roll down the window, to connect or ask or invite. For whatever reason, there is a knee-jerk need to deny any sadness, or dismiss it, or apologize for it. “I’m sorry,” people will say, wiping away their tears, as if their sadness is a violation of some tenet of propriety. Heaven forbid if any humanity is exposed.
So sometimes I am afraid to ask. Not sometimes; most times. I don’t want to appear weak. Asking for help is a hard pill to swallow.
I spent Saturday in Clearwater, Florida, with a group talking about intimacy and communication. (Yes, it is easier to talk about than to practice.) Here’s what I told the group.
If we don’t bring it with us, we’re not going to find it there. Which means intimacy–trust, vulnerability, authenticity, honesty–begins here.
With me. With this me.
I was raised in a religious environment that taught me to eradicate my messiness (to quash my sadness or grief or untidiness).
I now believe differently. I now know that we find and express acceptance, love and grace (the place where we can be fully human), in our messy, imperfect, and fully thorny selves. In other words: We can embrace this life–without any need to photoshop it.
To be human is to be vulnerable. I am capable of being wounded and cut and sad… which also means that I am capable of being kind and generous and present.
In such moments of heartache, I can have the courage to ask for a hand to hold.
In such moments of heartache, I can have the courage to hold a hand the needs to be held.
Robert Foster didn’t think twice about holding a hand that needed to be held. And he wasn’t posturing or amassing heavenly brownie points. He was doing what needed to be done.
Here’s the deal: we don’t need more remedies or advice. We need more touch. We become more human when we touch. Why? Because when we touch, we are seen. And when we are seen, we recognize that our value is not tied solely to our sorrow.
And we, you and I, will find no better lesson to learn from our beautiful canine friends, than this lesson of touch.
Reflecting more than a casual interest in this fascinating topic.
Yesterday, I referred to a recent visit to my doctor and the ‘system’ not being able to address two aspects at the same time, Vestibular Migraine and possible memory issues. Then later on, in response to a comment from Michelle of Dogkisses Blog, I owned up to having been advised that I may have early-onset Alzheimer’s Disease.
But very quickly friends responded to that ‘news’ by asking me if I had come across the information about coconut oil. So here are my findings about the possible curative effects of coconut oil and, clearly, I wanted to draw this to the attention of as many people as I can. Please feel free to republish this information; all I would ask is that you link back to the URL for this Post – thank you.
Let me take you to a website called Coconut Ketones and to a page on that website where there are a number of articles on the possible major healing effect of coconut oil. The primary article, written in 2008, starts thus,
WHAT IF THERE WAS A CURE FOR ALZHEIMER’S DISEASE AND NO ONE KNEW?
A Case Study by Dr. Mary Newport
July 22, 2008
There is a growing epidemic of obesity, type II diabetes, cardiovascular disease, and predictions that 15,000,000 people in the United States alone will have Alzheimer’s Disease by the year 2050.
In 2001, Dr. Richard L. Veech of the NIH, and others, published an article entitled, “Ketone bodies, potential therapeutic uses.”1 In 2003, George F. Cahill, Jr. and Richard Veech authored, “Ketoacids? Good Medicine?”2 and in 2004, Richard Veech published a review of the therapeutic implications of ketone bodies.3 These articles are not found in journals that the average physician would read, much less the lay public. Unless you are researching the topic, it is unlikely that you would ever randomly come across this information.
My husband Steve, age 58, has had progressive dementia for at least five years. He had an MRI in May 2008 showing a diffuse involutional change of the frontal and parietal lobes and moderate left-sided and severe right-sided amygdala and hippocampal atrophy with no ischemic change, which would support a clinical diagnosis of Alzheimer’s Disease. For non-medical people, this means that he has shrunken areas of the brain. Many days, often for several days in a row, he was in a fog; couldn’t find a spoon or remember how to get water out of the refrigerator. Some days were not so bad; he almost seemed like his former self, happy, with his unique sense of humor, creative, full of ideas. One day I would ask if a certain call came that I was expecting and he would say, “No.” Two days later he would remember the message from so-and-so from a couple of days earlier and what they said. Strange to have no short-term memory and yet the information was filed somewhere in his brain. My gut feeling is that diet has something to do with the fluctuation, but what. I knew that he was locked up in there somewhere, if only there was a key to open up the areas of his brain that he didn’t have access to.
The article goes on to show the amazing and positive differences that came about for Steve as a result of incorporating coconut oil and other dietary aspects. Please go here to view and download a pdf of the full article. But I will give you the closing paragraphs.
If you are using any type of hydrogenated vegetable oil or any oil with transfat, do not use any more and get rid of it! Extra virgin olive oil, butter and other natural, non-hydrogenated oils are okay to use along with the coconut oil. It is possible to use coconut oil in place of all other oils, however, since it contains no omega-3 fatty acids, it is very important to eat salmon twice a week or get enough omega-3 fatty acid from other rich sources such as fish oil capsules, flax meal, flax oil (not for cooking) or walnuts.
It is inconceivable that a potential dietary prevention and cure for Alzheimer’s disease, and other neurodegenerative diseases, has been out there for so many years, and yet has gone unnoticed. It is very likely that these diseases are becoming more prevalent due our current diet. The American diet has changed drastically from what it was before the 1950’s, when our parents and grandparents used lard and coconut oil to cook. Cardiovascular disease was rare at the beginning of the 20th century, and has skyrocketed, along with other devastating diseases, such as Alzheimer’s, diabetes type II, obesity, since mass produced hydrogenated vegetable oils containing trans fats were introduced into our diets and replaced these other natural fats. Sadly, the incidences of cardiovascular and other serious diseases are becoming more and more common among people in other areas of the world who have changed over from their indigenous foods to the “western” diet.
I plan to tell everyone I can and get this information to persons in positions to investigate this with the hope that Dr. Veech and other MCT oil and ketone body researchers get the funding they need. Feel free to make copies and pass this write-up on. If you have a loved one or a patient with Alzheimer’s or one of these other degenerative neurologic diseases, consider trying coconut oil. Dr. Veech suggests that, if possible, a videotape of the person before starting and at various points after starting the coconut oil would be very useful to document change. He suggests including segments of the persons face, speech and gait (walking). He also advises to have ketone bodies measured. What have you got to lose?
So in between eating spoonfulls of cold-compressed coconut oil, let me also give you some more information.
Here’s a video of Dr. Newport on CBN,
And here’s Part One of a video series all about Dr. Newport’s effective work on memory loss and Alzheimer’s with coconut oil.
Then I came across a paper delivered by Mary G. Enig, PhD on the Weston A. Price Foundation website. Here’s a flavour, pardon the pun, of this paper,
A New Look at Coconut Oil
Written by Mary G. Enig, PhD
January 1, 2000
Health and Nutritional Benefits from Coconut Oil: An Important Functional Food for the 21st Century
Presented at the AVOC Lauric Oils Symposium, Ho Chi Min City, Vietnam, 25 April 1996
Coconut oil has a unique role in the diet as an important physiologically functional food. The health and nutritional benefits that can be derived from consuming coconut oil have been recognized in many parts of the world for centuries. Although the advantage of regular consumption of coconut oil has been underappreciated by the consumer and producer alike for the recent two or three decades, its unique benefits should be compelling for the health minded consumer of today. A review of the diet/heart disease literature relevant to coconut oil clearly indicates that coconut oil is at worst neutral with respect to atherogenicity of fats and oils and, in fact, is likely to be a beneficial oil for prevention and treatment of some heart disease. Additionally, coconut oil provides a source of antimicrobial lipid for individuals with compromised immune systems and is a nonpromoting fat with respect to chemical carcinogenesis.
The long and detailed document ends, thus,
Among the critical foods and nutrition “buzz words” for the 21st Century is the term “functional foods.” Clearly coconut oil fits the designation of a very important functional food.
Last October, Dr. Mary Newport released a book, entitled Alzheimer’s Disease: What If There Was a Cure? The Story of Ketones.
Dr. Newport has a Blog which, despite not being added to since December 28th last, still has much interesting information.
Finally, I came across an Organic Coconut Oil Information website that opens up with,
Organic Coconut Oil is rich in vitamins and minerals and especially rich (60%+) in important fatty acids, the medium chain triglycerides (MCTs). It has been used by Asian and Pacific populations both as a source of dietary oil and in their traditional medical practices. Praised for its many and various healing properties, to a Pacific Islander, Coconut Oil is believed to be the cure of all illnesses and is so highly valued they refer to the coconut palm as “The Tree of Life.” Western modern science has only recently begun to uncover and understand the miracle healing value of the coconut.
Indeed, the website provides links to the health benefits of coconut oil for more than 17 other issues and, without lowering the tone too far, reminds me of an old joke.
Doctor, I’ve come to you about a bladder problem.
Sit down, Mr. Smith and tell me the details.
Well it’s just that I’m passing urine regularly every morning at 7am.
Mr. Smith, at your age that’s commendable having such a regular control, why would you regard that as a problem?
I’m not waking up until 8am!
Now where did I leave that last coconut!!